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Volume 123, Issue 1, Pages 25-28 (January 2003)


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Arch width after extraction and nonextraction treatment☆☆

Anthony A. Gianelly, DMD, PhD, MD

Received 1 March 2002; received in revised form 1 May 2002; accepted 1 May 2002.

Abstract 

An undocumented criticism of extraction treatment is that it results in narrower dental arches when compared with nonextraction therapy. Anterior and posterior arch widths of the maxillary and mandibular arches of 25 patients treated by 4 first-premolar extraction and 25 patients treated without extractions were measured on posttreatment study models and compared statistically to determine whether the dental arches were narrower after extraction treatment, to test the accuracy of this view. Measurements were made in the canine and the molar regions from the most labial aspect of the buccal surfaces of the canines and the molars. In both groups, anterior and posterior arch widths were the same except for the mandibular intercanine dimension, which was 0.94 mm larger (P < .01) in the extraction group. This indicates that extraction treatment does not result in narrower dental arches than nonextraction treatment. (Am J Orthod Dentofacial Orthop 2003;123:25-8)

Article Outline

Abstract

Material and methods

Results

Discussion

References

Copyright

To paraphrase the Bard of Avon: to extract, or not to extract: that is the question. This fundamental decision has spawned some intense debates in orthodontics.1 One issue is the effect of extraction on the buccal corridor, a recently introduced landmark that represents the space between the buccal surface of the dentition and the corresponding soft tissues with particular emphasis on the corners of the mouth.2, 3, 4 Presumably, the appearance of unesthetic black triangles at the corners of the mouth during smiling and dark shadows lateral to the buccal segments are expected sequellae of 4 first-premolar extraction treatment because this therapy narrows the width of the dental arches and shrinks the arches, resulting in a dentition that is too small to fill the oral slit during a smile.5

Although the assumed relationship has been accepted as conventional wisdom by some,6 its validity is questionable at best. Specifically, Johnson and Smith,2 in 1 of the few studies designed to evaluate this relationship in the refereed literature, used a “buccal corridor ratio,” defined as the maxillary intercanine width divided by the width of the mouth during a smile, to assess smile esthetics and found that the ratio was the same after both extraction and nonextraction treatment.

Also, the width of the dental arches, at least in the canine region, is generally not smaller after extraction treatment than after nonextraction therapy.7, 8 For example, the intercanine widths of both arches were statistically the same after extraction and nonextraction treatment of comparable groups of borderline subjects.7 And in patients whose long-term results were stable, the mandibular intercanine dimension increased more with extraction treatment9 than with nonextraction therapy.10, 11, 12, 13

Because a point of contention by those who espouse and support the buccal-corridor relationship is a narrowing of the dental arches, the purpose of this study was to compare anterior and posterior widths of the dental arches after both extraction and nonextraction therapy to determine whether extraction treatment results in narrower dental arches and, by inference, larger dark shadows in the buccal corridor.

Material and methods 

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The posttreatment study models of 25 patients who had 4 first-premolar extractions and 25 patients treated without extractions were randomly selected and measured. The only inclusion criterion was that all second molars were present and in alignment. Treatment was performed at a university clinic and directed by various instructors. All patients received fixed appliances; 49 were treated with the edgewise technique, and 1 with the tip-edge technique. In the extraction group, there were 12 Class I and 13 Class II malocclusions. In the nonextraction sample, there were 9 Class I, 13 Class II, and 3 Class III malocclusions.

With an electronic caliper (Digit-Cal, Browne and Sharpe, North Kingston, RI), the widths of the anterior and posterior parts of the maxillary and mandibular dental arches were measured in the canine and the molar regions from the most labial aspect of the buccal surfaces of those teeth. The caliper was placed at the best estimate of a right angle to the palatal suture in the maxillary arch and to a line bisecting the incisor segment in the mandibular arch. The recorded widths between the molars were the widest distances between the first or second molars. The widest part of the posterior part of the arch invariably was in the second molar region. Each distance was measured 3 times, and the average of the 3 values was used as the final measure.

Because arch width is ordinarily established by the mandibular arch, pretreatment widths between the mandibular canines and molars were recorded, as described, to establish that the arch widths of the mandibles of both samples were similar at the start of treatment. The treatment changes in the mandibular intercanine and intermolar dimensions were also determined. The pretreatment maxillary intercanine and intermolar dimensions were not measured because they vary in different malocclusions, and the distribution of malocclusions was not the same in each group.

The reproducibility of the measurements was evaluated by analyzing the differences between 10 double measurements of intercanine and intermolar distances, randomly selected and taken at different times. The error of measurement was assessed by Dahlberg's formula14: where D is the difference between duplicate measurements, and N is the number of double determinations. The errors were 0.22 mm for mandibular intercanine width, 0.50 mm for mandibular intermolar molar width, 0.26 mm for maxillary intercanine width, and 0.27 mm for maxillary intermolar width. Means and standard deviations were calculated, and a 2-tailed t test was used to determine statistically significant differences with P < .05.

Results 

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At the start of treatment, the mandibular intercanine and intermolar widths of both groups did not differ statistically (Table I).

Table I.

Pretreatment mandibular intercanine and intermolar arch widths: means and SD (mm)

Extraction (n = 25)
Nonextraction (n = 25)
Significance
3-331.47 ± 2.1031.24 ± 1.32NS
7-758.70 ± 3.0258.51 ± 1.67NS

3, Canine; 7, second molar; NS, not significant.

At the end of treatment, the arch widths of both groups were also statististically similar with 1 exception (Table II).
Table II.

Posttreatment maxillary and mandibular arch intercanine and intermolar widths: means and SD (mm)

Extraction (n = 25)
Nonextraction (n = 25)
Difference
Significance
3-3(L)32.87 ± 1.7431.93 ± 1.200.94.01
3-3(U)39.93 ± 2.0239.08 ± 1.640.83NS
7-7(L)57.54 ± 2.8058.38 ± 2.370.74NS
7-7(U)60.92 ± 2.0060.88 ± 2.910.04NS

3, Canine; 7, second molar; NS, not significant; L, maxillary; U, mandibular.

The average mandibular intercanine dimension was 0.94 mm larger in the extraction sample than in the nonextraction subjects. During treatment, the mandibular intercanine mean width was increased a statistically significant 1.39 mm in the patients treated with extractions, but the mean 0.69-mm increase in the nonextraction group was not statistically significant (Table III).
Table III.

Mandibular intercanine and intermolar width changes: means and SD (mm)

Extraction (n = 25)
Nonextraction (n = 25)
PretreatmentPosttreatmentDifferenceSignificancePretreatmentPosttreatmentDifferenceSignificance
3-331.48 ± 2.1032.87 ± 1.741.39.0231.24 ± 1.7431.93 ± 1.200.69NS (.06)
7-758.71 ± 3.0257.17 ± 2.191.54NS58.51 ± 1.7258.38 ± 2.37−0.13NS

3, Canine; 7, second molar; NS, not significant.

The mandibular intermolar widths of both the extraction and the nonextraction patients were essentially unchanged.

Discussion 

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To some advocates of nonextraction therapy, 4 first-premolar extraction treatment is synonymous with narrow dental arches with consequent unesthetic, large black triangles in the buccal corridor.5 This is not an accurate premise when describing treatment results obtained by fixed appliances. A comparison of posttreatment arch widths of patients treated by extraction and nonextraction procedures failed to identify the extraction patients because they had no characteristic narrow dental arches. In fact, the arches were approximately 1 mm wider in the mandibular canine area after extraction therapy, and this region establishes the width of the anterior part of the arches under normal conditions because the maxillary arch is conventionally fit to an idealized mandibular arch.

Although this finding might dissatisfy some nonextraction enthusiasts, particularly those who view extraction treatment negatively,5, 6 it is not a surprising observation if one examines the results of studies on posttreatment long-term stability in which mandibular incisor stability was acceptable. Specifically, the mandibular intercanine width increased 1.07 mm in an extraction sample.9 In contrast, in the nonextraction subjects, the increase in mandibular intercanine dimension was less than 1 mm in both Class I13 and Class II patients.10, 11, 12

In borderline patients, the long-term increase of the mandibular intercanine width in those treated by extraction therapy was 1.0 mm, whereas the increase in the nonextraction sample was only 0.5 mm.7 At the end of treatment, the intercanine widths of both groups were the same. Luppanapornlarp and Johnston8 evaluated the posttreatment and long-term results of treatment in clear-cut extraction and nonextraction patients and noted that the mandibular intercanine dimension of the extraction subjects was greater at all stages examined than the same parameter in the nonextraction patients. These data indicate that there is no systematic narrowing of the dental arches as a result of 4 first-premolar extraction treatment.

The increase in the mandibular intercanine width in this study in both extraction (1.4 mm) and nonextraction (0.69 mm) subjects compares with the previously cited increases recorded by others who evaluated patients treated by both strategies.7, 8, 15, 16 In these reports, a slightly larger increase occurred in those treated with extractions, possibly reflecting minor lateral movement as the canines are moved distally into the premolar sites. As an example, BeGole et al15 observed that the mandibular intercanine widths increased 1.58 mm in an extraction sample and 0.95 mm in nonextraction subjects. Uhde et al16 illustrated treatment changes in graph form in extraction and nonextraction subjects and demonstrated that the pretreatment mandibular intercanine widths were comparable in both groups, and, during treatment, a larger increase occurred in the extraction sample.

An unanticipated and surprising observation was that the intermolar widths of both groups of subjects were the same after treatment, because the usual expectation is that the posterior part of the arch becomes narrower as the molars move mesially into a narrower part of the arch during space closure. When including second molars in the assessment, this narrowing apparently does not occur. This observation also supports the view of Johnson and Smith2 that “typically, transverse arch width at any particular location in the buccal segments is maintained or slightly enlarged after extraction.” They also opined that the logic of those who believe that premolar extraction results in a reduction of the radius of the curve of the dental arch is faulty because the dental arch is not a circle and does not behave as a circle. Therefore, a reduction of the radius is not a necessary outcome of extracting a premolar on each side of the arch.

To some, maxillary arch width is a determinant of smile esthetics.4, 5, 6 Because maxillary arch width in the extraction and nonextraction subjects was the same, the effect of the 2 treatment strategies on smile esthetics would be similar. This conclusion is consistent with the results of a study that evaluated smile esthetics after both extraction and nonextraction treatment.2 There were no differences in esthetic scores between the groups. Interestingly, the investigators found that intercanine width, relative to the visible dentition, was wider in the extraction patients.

The labial surfaces of the canines and the molars were chosen as the measuring landmarks instead of the more customary cusp tips for several reasons: (1) to determine the widest possible widths of the arches, (2) to prevent confusion when selected cusps tips were not distinct, and (3) to avoid identifying a specific molar for measurement because in extraction treatment, as noted by Johnson and Smith,2 the first molars might have moved forward into a narrower part of the arch. For this reason, arch width measured between first molars would not adequately represent the width of the posterior aspect of the dental arches.

Changes in maxillary canine and molar widths were not recorded because the maxillary dentition does not customarily establish the widths of the dental arches. Also, these measurements can produce confusing information unless the groups compared are essentially identical at the outset. For instance, a pretreatment comparison of Class II Division 1 and Division 2 malocclusions with well-aligned mandibular arches and identical mandibular intercanine widths might show that the maxillary intercanine dimension is narrower in the Class II Division 1 malocclusion, particularly if the arch form in the overjet region is tapered. Under these conditions, the dimensional change resulting from treatment would be greater for the Class II Division 1 malocclusion even though the posttreatment intercanine distance in both examples would be the same because the mandibular intercanine distance is the same.

No patient in this study was treated with appliances specifically designed to increase mandibular arch width. The instability of excessive intercanine expansion (> 1-1.5 mm) in the mandibular arch17 is 1 reason that our customary treatment does not include these appliances.

In a more global sense, the contention that extraction treatment results in less esthetic smiles because of narrowing of the dental arches is, to an extent, similar to a once-presumed damaging effect of extraction therapy on the temporomandibular joint.18 In both instances, the consequences of extraction treatment are considered to be decidedly negative. The imputed injurious relationship between extraction treatment and temporomandibular disease has largely been discredited.19 The available data indicate that narrow dental arches are not the expected consequence of extraction treatment. Therefore, the esthetically compromising effect of narrow dental arches on smiles is not a systematic outcome of extraction treatment.

References 

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1. 1 Case CS. The question of extraction in orthodontia (reprint). Am J Orthod. 1964;50:660–691. Full-Text PDF (9617 KB) | CrossRef

2. 2 Johnson DK, Smith RJ. Smile esthetics after orthodontic treatment with and without extraction of four first premolars. Am J Orthod Dentofacial Orthop. 1995;108:162–167. Abstract | Full Text | Full-Text PDF (1162 KB) | CrossRef

3. 3 Dougherty HL. Clubs, quips, phrases, and hype: musings for the new millennium. Am J Orthod Dentofacial Orthop. 2000;117:586–588. Full Text | Full-Text PDF (42 KB) | CrossRef

4. 4 McNamara JA. Maxillary transverse deficiency. Am J Orthod Dentofacial Orthop. 2000;117:567–570. Full Text | Full-Text PDF (35 KB) | CrossRef

5. 5 Spahl TJ, Witzig JW. The clinical management of basic maxillofacial orthopedic appliances. Littleton (Mass): PSG Publishing Co; 1987;.

6. 6 Dierkes JM. The beauty of the face: an orthodontic perspective. J Am Dent Assoc. 1987;special number:89E–95E.

7. 7 Paquette DE, Beattie JR, Johnston LE. A long-term comparison of nonextraction and premolar extraction edgewise therapy in “borderline” Class II patients. Am J Orthod Dentofacial Orthop. 1992;102:1–14. Abstract | Full-Text PDF (1006 KB) | CrossRef

8. 8 Luppanapornlarp S, Johnston LE. The effects of premolar extraction: a long-term comparison of outcomes in “clear-cut” extraction and nonextraction Class II patients. Angle Orthod. 1993;63:257–272. MEDLINE

9. 9 Vaden JL, Harris EF, Ziegler Garner R. Relapse revisited. Am J Orthod Dentofacial Orthop. 1997;111:543–553. Abstract | Full Text | Full-Text PDF (1679 KB) | CrossRef

10. 10 Glenn G, Sinclair P, Alexander R. Nonextraction orthodontic therapy: posttreatment dental and skeletal stability. Am J Orthod Dentofacial Orthop. 1987;92:321–328. Abstract | Full-Text PDF (2311 KB) | CrossRef

11. 11 Moussa R, O'Reilly MT, Close JM. Long-term stability of rapid palatal expander and edgewise mechanotherapy. Am J Orthod Dentofacial Orthop. 1995;108:478–488. Abstract | Full Text | Full-Text PDF (898 KB) | CrossRef

12. 12 Yavari J, Shrout MK, Russell CM, Haas AJ, Hamilton EH. Relapse in Angle Class II Division 1 malocclusion treated by tandem mechanics without extraction of permanent teeth: a retrospective analysis. Am J Orthod Dentofacial Orthop. 2000;118:34–42. Abstract | Full Text | Full-Text PDF (40 KB) | CrossRef

13. 13 Azizi M, Shrout MK, Haas AJ, Russell CM, Hamilton EH. A retrospective study of Angle Class I malocclusions treated orthodontically without extractions using two palatal expansion methods. Am J Orthod Dentofacial Orthop. 1999;116:101–107. Abstract | Full Text | Full-Text PDF (31 KB) | CrossRef

14. 14 Dahlberg G. Statistical methods for medical and biological students. In: London: George Allen and Unwin Ltd; 1940;p. 122–132.

15. 15 BeGole EA, Fox DL, Sadowsky C. Analysis of change in arch form with premolar expansion. Am J Orthod Dentofacial Orthop. 1999;113:307–315. Abstract | Full Text | Full-Text PDF (183 KB) | CrossRef

16. 16 Uhde MD, Sadowsky C, BeGole EA. Long-term stability of dental relationships after orthodontic treatment. Angle Orthod. 1983;53:240–252. MEDLINE

17. 17 Burke SP, Silveira AM, Goldsmith LJ, Yancey JM, Stewart A, Scarfe WC. A meta-analysis of mandibular intercanine width in treatment and post retention. Angle Orthod. 1998;68:53–60. MEDLINE

18. 18 Farrar WB, McCarty WL. A clinical outline of temporomandibular joint diagnosis and treatment. In: Montgomery (Ala): Walker Printing Co; 1983;p. 84–85.

19. 19 McNamara JA, Seligman DA, Okeson JP. Orthodontics, occlusion and temporomandibular disorders. In:  McNamara JA,  Bruden WL editor. Orthodontics and dentofacial orthopedics. Ann Arbor (Mich): Needham Press; 2001;p. 519–544.

Professor and chairman, Department of Orthodontics, Boston University School of Dental Medicine. Boston, Mass

 Reprint requests to: Anthony A. Gianelly, 100 E Newton St, Boston, MA 02118-2392.

☆☆ 0889-5406/2003/$30.00 + 0

PII: S0889-5406(02)56908-0

doi:10.1067/mod.2003.57


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